The World Health Organization (WHO ) declared the outbreak of COVID-19 to be an international public health emergency. More than 4 million people are estimated to be contaminated worldwide.Thus ,underpins the address of health concern.
There are many coronaviruses ranging from the common cold to far more extreme viruses, including Extreme Severe Acute Respiratory Syndrome (SARS) and Middle East Respiratory Syndrome (MERS). These are viruses spread from animals to humans. Coronaviruses can cause lung infection (pneumonia), renal failure, and even death, in extreme cases. There is currently no COVID-19 vaccine.
Symptoms of COVID-19
Popular signs are common flu-like symptoms, such as fever , cough, respiratory issues, weakness, and muscle aches. Symptoms typically begin within 3–7 days of virus exposure, but in some cases , symptoms have taken up to 14 days.
People of any age can be contaminated. COVID-19 is mild for many (more than 80 % of the cases) with limited flu-like symptoms. Others did not exhibit signs or only rather mild symptoms, rather than a normal cold. The majority of people who contracted the virus did not need hospitalization. Nevertheless, COVID-19 was serious in up to 15 % of cases and essential disease in about 5 % of cases. The vast majority (approximately 98 percent) of infected individuals have lived to date.
Older individuals and persons with pre-existing medical conditions (such as diabetes , heart disease and asthma) are more vulnerable to severe COVID-19 disease. When people with diabetes develop a viral infection, fluctuations in blood glucose and possible diabetes complications can make it harder to treat. :
Covid-19 and diabetes have a bidirectional relationship. Diabetes, on the one hand, is associated with an elevated risk of severe Covid-19. New diabetes and significant metabolic complications of current diabetes in patients with Covid-19, on the other hand, have been reported.
SARS & non SARS Patients:
There are also many precedents of a ketosis-prone viral cause of diabetes, including certain coronaviruses that bind to ACE2 receptors. More frequent incidences of fasting glycemia and acute diabetes among SARS coronavirus-1 patients have been recorded than among non-SARS pneumonia patients.These observations support a hypothesis, in addition to the well-recognized stress response associated with a severe illness, of a potential diabetogenic effect of Covid-19. However, whether glucose metabolism changes that occur when a severe Covid-19 suddenly occur or do persist when the infection resolves are not clear